For your skin
The ultimate "cell recharger." It boosts your skin's cellular energy to speed up repair and protect against aging at the DNA level. It's the new high-tech standard for longevity skincare.
Want the science? Keep reading ↓Mechanism of action
Essential coenzyme for cellular energy (ATP) production and DNA repair; serves as a substrate for sirtuins and PARPs to regulate cellular senescence and maintain genomic stability in skin cells.
Why we tier this anecdotal
2 cited papers across 2 countries. Most of what's cited here is mechanism-level or in-vitro work. We track this as Anecdotal until controlled clinical trials accumulate.
Cited research
Ministrini S, Liberale L, Erle HE, Percoco G, Tfayli A, Assi A, Kapitonov I, Greiner I, Camici GG, A Liposomal Formulation Enhances the Anti-Senescence Properties of Nicotinamide Adenine-Dinucleotide (NAD+) in Endothelial Cells and Keratinocytes, Current Issues in Molecular Biology 2025 — in-vitro: liposomal NAD+ reduced senescent cells in human keratinocytes and endothelial cells and increased skin penetration ~30% vs free NAD+ (cell-based mechanism)
Kang S, Park J, Cheng Z, Ye S, Jun SH, Kang NG, Novel Approach to Skin Anti-Aging: Boosting Pharmacological Effects of Exogenous Nicotinamide Adenine Dinucleotide (NAD+) by Synergistic Inhibition of CD38 Expression, Cells 2024 — in-vitro: exogenous NAD+ on human fibroblasts protected against UV-induced and intrinsic aging via sirtuin/autophagy pathways; effect amplified by CD38 inhibition (cell-based mechanism)
Sources: PubMed · KCI · J-Stage · CNKI · Wanfang · SFD · MFDS · Cochrane · SCCS · CIR. Every entry points to a specific document. See methodology for what each outcome label means.